Stanford: The ulcer-causing bacterium Helicobacter pylori can directly
interact with stomach stem cells, causing the cells to divide more
rapidly, according to a new study by researchers at the Stanford University School of Medicine. The increased cell division was observed in mice, but the findings could explain why H. pylori is a risk factor for gastric cancer in humans, the researchers said.
They used 3-D microscopy to identified colonies of the bacteria deep within human stomach glands, where stem cells and precursor cells that replenish the stomach’s lining reside.
They used 3-D microscopy to identified colonies of the bacteria deep within human stomach glands, where stem cells and precursor cells that replenish the stomach’s lining reside.