Monday, January 19, 2015

Earlier tPA Treatment for Ischemic Stroke

Mass general Hospital. US: Tissue plasminogen activator (tPA) is the only approved drug for ischemic stroke, those 85 percent of strokes caused by thrombosis in the brain. After four and a half hours from stroke onset, the risk of brain hemorrhaging in response to tPA increases. Guidelines recommend that patients entering the hospital should receive tPA within 4.5 hours from the time they were last known to be well, and that it should be given within 60 minutes of arriving at the hospital (the “door-to-needle,” or DTN, time), but many patients do not arrive in time and many emergency departments cannot marshal resources quickly enough.

Community hospitals also often lack rapid access to a neurologist and will not administer tPA without one.
As a result, less than a third of eligible stroke patients receive tPA within the recommended DTN time1, almost 20 years after its introduction and despite strong evidence that rapid treatment with tPA translates into better patient outcomes.2 At Massachusetts General Hospital, Lee Schwamm, MD, Director of the Mass General Stroke Services within the Institute for Heart, Vascular and Stroke Care, Director of the Partners Telestroke Center and Vice Chairman of the Department of Neurology, aims to increase the number of patients treated with tPA, accelerate the DTN time, and promote telestroke programs that connect neurologists with community hospitals, effectively converting them into high-performance stroke centers.

Target: Stroke Interventions Improve Clinical Outcomes

Mass General was one of 1,030 Get With the Guidelines-Stroke (GWTG-Stroke) hospitals across the United States participating in Target: Stroke, a quality improvement program based on the rationale that rapid treatment with tPA translates into better patient outcomes.3 The primary goal of the quality improvement program was to ensure that at least 50 percent of patients receiving tPA in the U.S. were receiving it within 60 minutes of hospital arrival. The study, published in JAMA in April 2013,4 followed 27,319 ischemic stroke patients who received tPA during a pre-intervention period between 2003 and 2009, and 43,850 during a post-intervention period from 2010 to 2013.
The intervention entailed implementing 10 evidence-based strategies, which included pre-notification of hospitals by EMTs to activate the stroke team; rapid acquisition and interpretation of brain imaging; premixing tPA for high-likelihood candidates; and a stroke team-based approach. The proportion of patients receiving tPA in 60 minutes or less increased by 1.36 percent per year in the pre-intervention period and by 6.2 percent in the post-intervention period, and clinical outcomes (mortality, discharge to home, ambulatory status, symptomatic intracranial hemorrhage) all improved post-intervention. Importantly, the accelerated pace of improvement in the post-intervention period shortened the projected time to reach the objective of the intervention by 10 years. By the fourth quarter of 2013, the objective had been surpassed, with 53.3 percent of tPA patients being treated in 60 minutes or less, a significant rise from the 26.5 percent who met the time target goal in early 2003.

To determine whether clinical improvements were attributable to earlier tPA administration or to general improvements in care for all stroke patients, the study compared outcomes of stroke patients at the same GWTG-Stroke hospitals who did not receive tPA. In one example, reduction of in-hospital mortality improved for non-tPA patients by 4 percent, but improved 11 percent for tPA patients. “This is the first study to show that in real-world practice, tPA works best the faster you give it, and that faster treatment saves lives and reduces bleeding complications,” Dr. Schwamm said.
Distinguishing Stroke Mimics From Strokes

One unintended consequence of the effort to improve DTN time for tPA might be an increase in false positives, given the rush to diagnose and treat patients. Seizures, epilepsy, migraines, cardiovascular morbidities and diabetes can all mimic classic stroke symptoms: facial weakness, limb weakness, speech difficulty and altered level of consciousness. While treating such stroke mimics (SMs) with tPA generally does not harm patients, unless they have damaged tissue that could hemorrhage, Dr. Schwamm’s team saw this as a costly and avoidable drain on resources. In connection with telestroke programs that use telecommunication technologies to evaluate stroke patients remotely, Dr. Schwamm determined that SM patients were approximately 10 years younger than patients with true stroke syndromes and were less likely to have a history of hypertension, atrial fibrillation and heart failure. Two striking indicators of SM are an absence of facial weakness and presence of a seizure disorder. His team developed a prediction rule based on six variables (age, history of atrial fibrillation, hypertension, seizure, facial weakness on exam and moderate to severe stroke as measured by the NIH Stroke Scale) that help telestroke consults differentiate between SMs and patients with probable ischemic stroke.5 Dr. Schwamm’s ultimate goal is to have the model for stroke medicine emulate more closely that of cardiac emergencies, with its coordinated and specialized teams. Despite the absence of new drugs, better stroke treatment and outcomes are achievable with existing medications. It does not require a new invention, a new paradigm or new costs, but rather an intensive effort to prioritize more rapid tPA administration.
References
1 Jauch, E.C., J. L. Save, H. P. Adams Jr., et al. American Heart Association Stroke Council; Council on Cardiovascular Nursing; Council on Peripheral Vascular Disease; Council on Clinical Cardiology. ”Guidelines for the early management of patients with acute ischemic stroke: a guideline for healthcare professionals from the American Heart Association/American Stroke Association.” Stroke. 2013;44(3):870-947.
2 Ibid
3 Fonarow, G. C., E. E. Smith, J. L. Saver, et al. “Timeliness of tissue-type plasminogen activator therapy in acute ischemic stroke: patient characteristics, hospital factors, and outcomes associated with door-to-needle times within 60 minutes.” Circulation. 2011;123(7):750-758.
4 Fonarow, G. C., X. Zhao, E. E. Smith et al. 2014. “Door-to-Needle Times for Tissue Plasminogen Activator Administration and Clinical Outcomes in Acute Ischemic Stroke before and after a Quality Improvement Initiative.” JAMA 311 (16): 1632–40. doi:10.1001/jama.2014.3203. 5 Ali, Syed F., Anand Viswanathan, Aneesh B. Singhal, Natalia S. Rost, Pamela G. Forducey, Lawrence W. Davis, Joseph Schindler, et al. 2014. “The TeleStroke Mimic (TM)-Score: A Prediction Rule for Identifying Stroke Mimics Evaluated in a Telestroke Network.” Journal of the American Heart Association 3 (3): e000838. doi:10.1161/JAHA.114.000838.
Contributor
Lee H. Schwamm, MD
  • Executive Vice Chairman, Neurology
  • Director, Stroke Services and Partners TeleStroke Center